El virus de la immunodeficiència felina (FIV o VIH felí) pertany al gènere .. Alberto Fernández Lommen et al; Infección por Virus de la inmunodeficiencia felina. Many translated example sentences containing “virus inmunodeficiencia felina” – English-Spanish dictionary and search engine for English translations. Los virus de la inmunodeficiencia felina y de la leucemia linfoide en gatos son muy similares en cuanto a las lesiones y síntomas que producen, por lo que se.
|Published (Last):||19 April 2017|
|PDF File Size:||19.40 Mb|
|ePub File Size:||20.79 Mb|
|Price:||Free* [*Free Regsitration Required]|
Virus de la inmunodeficiencia felina VIF: Directory of Open Access Journals Sweden. Prevalencia de leucemia viral felinainmunodeficiencia viral felina y dirofilariosis felina en gatos refugiados en un albergue de animales en Maracaibo, Venezuela.
Full Text Available The gradual increment of the feline population in Colombia and some countries is associated with presence of diseases that care produce animal health risk. The animals were submitted to a physical and laboratory examination the population studied were 30 females and 30 males most of them minor of 2 years.
Blood samples from cats with symptoms compatible with FIV or FeLV infection, collected between and were analyzed by serology immunochromatography, IA and by hem Feline immunodeficiency virus FIV is a lentivirus associated with immunologic disorders in domestic cats. Due to the high genetic variability of FIVfive subtypes A to E have been identified and diversity within each subtype is also frequent.
The study of the genetic diversity can aid the understanding the pathogenesis and epidemiology of the disease. The sequencing of bp of the gag gene from 23 samples was performed and the results were analyzed using the Tamura-Nei nucleotidic substitution method. The phylogenetic analysis showed that all viruses belong to. Two methods for proviral DNA extraction and two pairs of primers were evaluated. The method for amplification was standardized and its specificity was determined by the restriction endonucleases digestion of PCR products.
Antibodies specific to FIV were determined using a commercial kit. Thirty heparinized blood samples obtained from cats with and without clinical signs consistent with FIV i The occurrence of the feline immunodeficiency virus FIV and feline leukemia virus FeLV was investigated in domestic cats from two shelters of Belo Horizonte.
For FeLV tests, 13 cats Pathogenesis of oral FIV infection. Full Text Available Feline immunodeficiency virus FIV is the feline analogue of human immunodeficiency virus HIV and features many hallmarks of HIV infection and pathogenesis, including the development of concurrent oral lesions. While it is accepted that FIV is primarily transmitted by biting, few studies have evaluated FIV oral infection kinetics and transmission mechanisms over the last 20 years. Modern quantitative analyses applied to natural FIV oral infection could significantly further our understanding of lentiviral oral disease and transmission.
We therefore characterized FIV salivary viral kinetics and antibody secretions to more fully document oral viral pathogenesis. Our results demonstrate that: Collectively, these results suggest that oral lymphoid tissues serve as a site for enhanced FIV replication, resulting in accumulation of FIV particles and FIV -infected cells in saliva.
Based upon these findings, we propose a model of oral FIV pathogenesis. Inmunodeficiencias en la infancia. Primary or secondary immunodeficiencies are pathological states, where a partial or total defect is observed in one or various of the components of the immune system.
A detailed questionnaire, physical examination and complementary tests, allow to make a positive diagnosis of an immunodeficiency. The chronic or recurrent infection is the most common clinical sign. The most frequent causes of immunodeficiencies, as well as of other pathological states conditioning the harmless infections in immunity, are related. The differential diagnosis makes possible to establish an early therapeutics in order to prolongue the life of the immunodeficient patient to prevent unnecessary manipulations or expenses concerning the infected patient without deteriorating immunity.
The aim of this study was to determine if saliva is a suitable diagnostic specimen using the same well characterized feline cohort. FIV infection status of these cats had been determined inmundoeficiencia using a combination of serology, polymerase chain reaction PCR testing and virus isolation.
Como todos los retrovi The results showed that The clinical evaluation showed that In addition, the vast majority The incidence of FIV infection was higher in males The results suggest that certain characteristics such as gender, health status and lifestyle may be associated with the risk of being infected with FIV in the population of cats studied.
Inmnodeficiencia study challenges the commonly held view that the feline immunodeficiency virus FIV infection status of FIV -vaccinated cats cannot be determined using point-of-care antibody test kits due to indistinguishable antibody production in FIV -vaccinated and naturally FIV -infected cats.
These results will potentially change the way veterinary practitioners screen for FIV in jurisdictions where FIV vaccination is practiced, especially in shelter scenarios where the feasibility inmunodeficiencua mass screening is impacted by the cost of testing. Published by Elsevier Ltd.
Ltd, Daejeon Korea, Republic of. Therefore, in addition to the normal control action that closes the main feedwater valves, a safety injection signal rapidly closes all Feed water Control Valve FCV s and Feedwater Isolation Valve FIV s, trips the main feedwater pumps, and closes the feedwater pump discharge valves. However, in a certain plant, the Eflina are not automated.
If the FIVs could not be credited, the trip of main feedwater pumps can be act as back up protection measures for the single failure of FVCs.
Virus de la immunodeficiència felina – Viquipèdia, l’enciclopèdia lliure
In that case, un isolated feedwater which is contained in the pipe between the main feedwater pump and the upstream of the FCV might be flash and be supplied to the broken steam generator. The containment integrity was studied for this case.
FIV Ple subtype composition may influence disease outcome in African lions. Feline immunodeficiency virus FIV infects domestic cats and at least 20 additional species of non-domestic felids throughout the world. Less is known about the immunological and pathological influence of FIV in other felid species although multiple distinct strains of FIV circulate in natural populations. Herein, the relative distribution of these subtypes in the population are surveyed and, combined with observed differences in lion mortality due to secondary infections based on FIV Ple subtypes, the data suggest that FIV Ple subtypes may have different patterns of pathogenicity and transmissibility among wild lion populations.
Pharmacological inhibition of feline immunodeficiency virus FIV. Feline immunodeficiency virus FIV is a member of the retroviridae family of viruses and causes an acquired immunodeficiency syndrome AIDS in domestic and non-domestic cats worldwide. Genome organization of FIV and clinical characteristics of the disease caused by the virus are similar to those of human immunodeficiency virus HIV.
Both viruses infect T lymphocytes, monocytes and macrophages, and their replication cycle in infected cells is analogous. Due to marked similarity in genomic organization, virus structure, virus replication and disease pathogenesis of FIV and HIV, infection of cats with FIV is a useful tool to study and develop novel drugs and vaccines for HIV. Anti-retroviral drugs studied extensively in HIV infection have targeted different steps of the virus replication cycle: Despite much success of anti-retroviral therapy slowing disease progression in people, similar therapy has not been thoroughly investigated in cats.
In this article we review current pharmacological approaches and novel targets for anti-lentiviral therapy, and critically assess potentially suitable applications against FIV infection in cats. Full Text Available One of the major obstacles that must be overcome in the design of effective lentiviral vaccines is the ability of lentiviruses to evolve in order to escape from neutralizing antibodies. The primary target for neutralizing antibodies is the highly variable viral envelope glycoprotein Env, a glycoprotein that is essential for viral entry and comprises both variable and conserved regions.
As a result of the complex trimeric nature of Env, there is steric hindrance of conserved epitopes required for receptor binding so that these are not accessible to antibodies. Instead, the humoral response is targeted towards decoy immunodominant epitopes on variable domains such as inmunoreficiencia third hypervariable loop V3 of Env.
For feline immunodeficiency virus FIVas well as the related human immunodeficiency virus-1 HIV-1, little is known about the factors that lead to the development of broadly neutralizing antibodies. In cats infected with FIV and patients infected with HIV-1, only rarely are plasma samples found that contain antibodies capable of neutralizing isolates from other clades.
We ask whether broadly neutralizing antibodies are induced by FIV infection and discuss the comparative value of studies of neutralizing inmunodefidiencia in FIV infection for the development of more effective vaccine strategies against lentiviral infections in general, including HIV Inmunodeficiencia y autoinmunidad constituyen 2 polos de la respuesta inmunitaria que pueden interrelacionarse con cierta frecuencia.
IFA modified in our laboratory, was compared with two other immunoassays, western blot WB and a sandwich immunochromatographic commercial kit SI, and also with a direct polymerase chain reaction PCR method that detects proviral DNA. IFA showed to be a test with high sensitivity and specificity, and could be useful as a diagnostic tool in epidemiological studies. The presence of a low percentage of fellina with non-specific reactivity in the IFA could be resolved with further testing or use inmunodefuciencia an alternative method.
The molecular biology of feline immunodeficiency virus FIV. It is highly prevalent in several endangered feline species. In recent years there has been sa significant increase in interest in FIVin part to exploit this, but also because of the potential it has as a human gene therapy vector. Though much less studied than HIV there are many parallels in the replication of the two viruses, but also important differences and, despite their likely common origin, the viruses have in some cases used alternative strategies to overcome similar problems.
Recent advances in understanding the structure and function of FIV RNA and proteins and their interactions has enhanced our knowledge of FIV replication significantly, however, there are still many gaps. This review summarizes our current knowledge of FIV molecular biology and its similarities with, and differences from, other lentiviruses.
Otro Grado en Medicina. Uso da fluoxetina no tratamento da tricotilomania felina. Full Text Available Feline Immunodeficiency Virus is a worldwide infection and is considered a significant pathogen. The diagnosis of FIV infections is mainly based on commercially available rapid tests that are highly expensive in Brazil, hence it is rarely performed in the country.
Furthermore, lentiviruses grow slowly and poorly in tissue cultures, making the production of viral antigen by classic means and thus the establishment of FIV immunodiagnosis impracticable. In inmynodeficiencia to deal with this, recombinant DNA techniques were adopted to produce the protein p24, a viral capsid antigen.
The protein’s reactivity evaluation analyzed by Western blot indicated that this recombinant antigen can be a useful tool for the immunodiagnostic of FIV infections.
inmunodeficiencia felina fiv: Topics by
Antibody testing based on individual risk assessments is recommended to determine feline immunodeficiency virus FIV status, but ELISA and Western blot tests cannot distinguish between anti- FIV antibodies produced in response to natural infection and those produced in response to FIV vaccination.
Characterization of regionally associated feline immunodeficiency virus FIV in bobcats Lynx rufus. However, recent studies have detected an FIV strain infecting both bobcats Lynx rufus and pumas Puma concolor in California and Florida.
Partial viral sequences from five Florida bobcats cluster with previously published sequences from Florida panthers. We did not detect FIV in Colorado bobcats. Feline immunodeficiency virus FIVa feline lentivirus related to HIV, causes immune dysfunction in domestic and wild cats.
Rnas inhibidores frente al virus de la inmunodeficiencia humana. En la presente tesis se describe la generaci??
Este virus es el agente causal del S??
Virus de la immunodeficiència felina
El virus presenta una alternancia en cuanto al tipo de material gen?? Full Text Available Feline immunodeficiency virus FIV is a naturally-occurring retrovirus that infects domestic and non-domestic feline species, producing progressive immune depletion that results in an acquired immunodeficiency syndrome AIDS.
Much has been learned about FIV since it was first described inparticularly in regard to its application as a model to study the closely related lentivirus, human immunodeficiency virus HIV. In particular, FIV and HIV share remarkable structure and sequence organization, utilize parallel modes of receptor-mediated entry, and result in a similar spectrum of immunodeficiency-related diseases due to analogous modes of immune dysfunction.
This review summarizes current knowledge of FIV infection kinetics and the mechanisms of immune dysfunction in relation to opportunistic disease, specifically in regard to studying HIV pathogenesis.
Furthermore, we present data that highlight changes in the oral microbiota and oral immune system during FIV infection, and outline the potential for the feline model of oral AIDS manifestations to elucidate pathogenic mechanisms of HIV-induced oral disease.